Clinical nutrition ESPEN · 2025
Superior mesenteric artery syndrome patients with hypoglycaemic brain injury can Be treated with nutritional therapy. A case report.
СтатьяКлинический случайНазвание и аннотация: Перевод в очереди
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Murong Min, Liang Weifen, Peng Suliu, Jiang Mengliu, Xiao Guizhen
Аннотация
BACKGROUND: Nutritional therapy is a primary intervention in treating superior mesenteric artery syndrome (SMAS). However, the role of nutritional therapy in patients with SMAS who are in a long-term catabolic phase with hypoglycaemia and serious complications associated with hypoglycaemia due to insufficient energy reserves has never been reported.
CASE REPORT: We present a 22-year-old male with SMAS who presented with a history of recurrent vomiting of gastric contents; the primary clinical manifestation is a history of the ventriculoperitoneal shunt (VP-shunts) surgery for obstructive hydrocephalus with postoperative cryptococcal meningitis. The course is repetitive and unstable and does not heal long. Furthermore, the patient experienced severe weight loss and hypoglycemic events, which eventually resulted in a hypoglycemic brain injury. In this case, individualized nutritional therapy successfully alleviated the patient's clinical symptoms, improved nutritional status, and prevented recurrent episodes of hypoglycemia and serious complications.
DISCUSSION: Our results indicate that nutritional therapy not only alleviates clinical symptoms and enhances nutritional status in SMAS patients but also prevents life-threatening complications associated with the condition. However, these findings require confirmation in a larger patient cohort.
CONCLUSION: Nutritional therapy is effective in reducing symptoms and improving nutritional status in SMAS patients with hypoglycaemic brain injury, as reported for the first time. The case also highlights the susceptibility of SMAS patients to hypoglycemic events and the prime time for nutritional therapy.
CASE REPORT: We present a 22-year-old male with SMAS who presented with a history of recurrent vomiting of gastric contents; the primary clinical manifestation is a history of the ventriculoperitoneal shunt (VP-shunts) surgery for obstructive hydrocephalus with postoperative cryptococcal meningitis. The course is repetitive and unstable and does not heal long. Furthermore, the patient experienced severe weight loss and hypoglycemic events, which eventually resulted in a hypoglycemic brain injury. In this case, individualized nutritional therapy successfully alleviated the patient's clinical symptoms, improved nutritional status, and prevented recurrent episodes of hypoglycemia and serious complications.
DISCUSSION: Our results indicate that nutritional therapy not only alleviates clinical symptoms and enhances nutritional status in SMAS patients but also prevents life-threatening complications associated with the condition. However, these findings require confirmation in a larger patient cohort.
CONCLUSION: Nutritional therapy is effective in reducing symptoms and improving nutritional status in SMAS patients with hypoglycaemic brain injury, as reported for the first time. The case also highlights the susceptibility of SMAS patients to hypoglycemic events and the prime time for nutritional therapy.