JACC. Case reports · 2026
Complete Heart Block Related to Bicuspid Aortic Valve Calcification in a Triathlete.
Клинический случайСтатьяНазвание и аннотация: Перевод в очереди
Обработка статьи · этапы, ошибки и управление
Загрузка состояния…
Ошибки и ограничения
История · было → стало
Состояние обновляется каждые 5 секунд, пока блок открыт. Процент показывается только там, где известен объём работы. Пауза останавливает продолжение этапа; текущий запрос к источнику может завершиться.
Chandra Abhishek, Downey Michael C, Olson Matthew D, Harris Kevin M
Аннотация
BACKGROUND: While athletic patients may exhibit bradycardia, significant exertional symptoms in the setting of a bicuspid aortic valve (BAV) warrant further investigation.
CASE SUMMARY: A 36-year-old male triathlete presented with progressive exercise intolerance, fatigue, and bradycardia. Electrocardiogram demonstrated complete atrioventricular block (junctional ventricular escape: 52 beats/min). Echocardiography revealed a moderately calcified BAV with mild to moderate stenosis and preserved ventricular function. Cardiac imaging demonstrated a Sievers type 0 valve with asymmetric extravalvular calcification extending to the atrioventricular node and perimembranous septum near the penetrating His bundle, providing a mechanistic explanation for conduction block. A dual-chamber left bundle branch pacemaker was implanted, and the patient shortly returned to baseline performance.
DISCUSSION: This athlete's case highlights a rare presentation of complete heart block secondary to BAV-associated calcification, demonstrating its conduction implications.
TAKE-HOME MESSAGE: Bradycardia and decreased exercise tolerance in athletes warrant thorough evaluation for rare structural causes of conduction disease.
CASE SUMMARY: A 36-year-old male triathlete presented with progressive exercise intolerance, fatigue, and bradycardia. Electrocardiogram demonstrated complete atrioventricular block (junctional ventricular escape: 52 beats/min). Echocardiography revealed a moderately calcified BAV with mild to moderate stenosis and preserved ventricular function. Cardiac imaging demonstrated a Sievers type 0 valve with asymmetric extravalvular calcification extending to the atrioventricular node and perimembranous septum near the penetrating His bundle, providing a mechanistic explanation for conduction block. A dual-chamber left bundle branch pacemaker was implanted, and the patient shortly returned to baseline performance.
DISCUSSION: This athlete's case highlights a rare presentation of complete heart block secondary to BAV-associated calcification, demonstrating its conduction implications.
TAKE-HOME MESSAGE: Bradycardia and decreased exercise tolerance in athletes warrant thorough evaluation for rare structural causes of conduction disease.