Cancer cell · 2023
Genomic mapping of metastatic organotropism in lung adenocarcinoma.
СтатьяПоддержка правительства СШАПоддержка NIH: внешняя программаНазвание и аннотация: Перевод в очереди
Обработка статьи · этапы, ошибки и управление
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Ошибки и ограничения
История · было → стало
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Lengel Harry B, Mastrogiacomo Brooke, Connolly James G, Tan Kay See, Liu Yuan, Fick Cameron N, Dunne Elizabeth G, He Di, Lankadasari Manendra B, Satravada Baby Anusha, Sun Yichao, Kundra Ritika, Fong Chris, Smith Shaleigh, Riely Gregory J, Rudin Charles M, Gomez Daniel R, Solit David B, Berger Michael F, Li Bob T, Mayo Marty W, Matei Irina, Lyden David C, Adusumilli Prasad S, Schultz Nikolaus, Sanchez-Vega Francisco, Jones David R
Аннотация
We analyzed 2,532 lung adenocarcinomas (LUAD) to identify the clinicopathological and genomic features associated with metastasis, metastatic burden, organotropism, and metastasis-free survival. Patients who develop metastasis are younger and male, with primary tumors enriched in micropapillary or solid histological subtypes and with a higher mutational burden, chromosomal instability, and fraction of genome doublings. Inactivation of TP53, SMARCA4, and CDKN2A are correlated with a site-specific shorter time to metastasis. The APOBEC mutational signature is more prevalent among metastases, particularly liver lesions. Analyses of matched specimens show that oncogenic and actionable alterations are frequently shared between primary tumors and metastases, whereas copy number alterations of unknown significance are more often private to metastases. Only 4% of metastases harbor therapeutically actionable alterations undetected in their matched primaries. Key clinicopathological and genomic alterations in our cohort were externally validated. In summary, our analysis highlights the complexity of clinicopathological features and tumor genomics in LUAD organotropism.