Materials today. Bio · 2026
Injectable polysaccharide hydrogel microspheres integrated with BPNS-ZnO nanocomposites restore redox homeostasis and protect cartilage in osteoarthritis.
СтатьяНазвание и аннотация: Перевод в очереди
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Lei Yuqing, Ma Tao, Lin Xiaolong, Sun Yuting, Liu Chenlong, Yao Jiapei, Wu Jingwei, Li Junyang, Tu Xiaoshuang, Pang Bo, Lu Zhuoyi, Li Dong, Zhang Ling, Shang Jingjing, Nong Luming, Zhou Xindie
Аннотация
Osteoarthritis (OA) is a progressive whole-joint disease driven by a persistent inflammatory-oxidative microenvironment that fuels cartilage destruction and osteochondral remodeling, while effective disease-modifying interventions remain scarce. Here, an injectable microsphere platform (HM@BPZn) is engineered by incorporating electronically coupled BPNS-ZnO hybrid nanosheets into a dual-polysaccharide network of methacrylated hyaluronic acid and gallic acid-grafted chondroitin sulfate. Interfacial electronic redistribution within the BPNS-ZnO hybrids enables active modulation of the pathological joint microenvironment. HM@BPZn suppresses reactive oxygen species accumulation and NF-κB signaling in macrophages, reducing inflammatory mediator production and promoting pro-resolving polarization. In inflamed chondrocytes, HM@BPZn activates the Keap1-Nrf2 pathway, upregulates antioxidant effectors including HO-1 and NQO1, and restores matrix homeostasis by suppressing catabolic markers while enhancing anabolic and lubrication-associated phenotypes. Transcriptomic profiling further verifies coordinated rewiring of inflammatory and antioxidant signaling networks. In an ACLT-induced OA model, intra-articular delivery of HM@BPZn markedly mitigates synovitis, preserves proteoglycan-rich cartilage, and attenuates pathological subchondral bone remodeling and osteophyte formation. These findings establish HM@BPZn as an injectable microenvironment-regulating platform for disease-modifying OA treatment.