Nature communications · 2015
NELL-1 in the treatment of osteoporotic bone loss.
СтатьяПоддержка NIH: внешняя программаНазвание и аннотация: Перевод в очереди
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James Aaron W, Shen Jia, Zhang Xinli, Asatrian Greg, Goyal Raghav, Kwak Jin H, Jiang Lin, Bengs Benjamin, Culiat Cymbeline T, Turner A Simon, Seim Iii Howard B, Wu Benjamin M, Lyons Karen, Adams John S, Ting Kang, Soo Chia
Аннотация
NELL-1 is a secreted, osteoinductive protein whose expression rheostatically controls skeletal ossification. Overexpression of NELL-1 results in craniosynostosis in humans and mice, whereas lack of Nell-1 expression is associated with skeletal undermineralization. Here we show that Nell-1-haploinsufficient mice have normal skeletal development but undergo age-related osteoporosis, characterized by a reduction in osteoblast:osteoclast (OB:OC) ratio and increased bone fragility. Recombinant NELL-1 binds to integrin β1 and consequently induces Wnt/β-catenin signalling, associated with increased OB differentiation and inhibition of OC-directed bone resorption. Systemic delivery of NELL-1 to mice with gonadectomy-induced osteoporosis results in improved bone mineral density. When extended to a large animal model, local delivery of NELL-1 to osteoporotic sheep spine leads to significant increase in bone formation. Altogether, these findings suggest that NELL-1 deficiency plays a role in osteoporosis and demonstrate the potential utility of NELL-1 as a combination anabolic/antiosteoclastic therapeutic for bone loss.