Nature communications · 2021
A deep learning approach to identify gene targets of a therapeutic for human splicing disorders.
СтатьяПоддержка NIH: внешняя программаПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
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Gao Dadi, Morini Elisabetta, Salani Monica, Krauson Aram J, Chekuri Anil, Sharma Neeraj, Ragavendran Ashok, Erdin Serkan, Logan Emily M, Li Wencheng, Dakka Amal, Narasimhan Jana, Zhao Xin, Naryshkin Nikolai, Trotta Christopher R, Effenberger Kerstin A, Woll Matthew G, Gabbeta Vijayalakshmi, Karp Gary, Yu Yong, Johnson Graham, Paquette William D, Cutting Garry R, Talkowski Michael E, Slaugenhaupt Susan A
Аннотация
Pre-mRNA splicing is a key controller of human gene expression. Disturbances in splicing due to mutation lead to dysregulated protein expression and contribute to a substantial fraction of human disease. Several classes of splicing modulator compounds (SMCs) have been recently identified and establish that pre-mRNA splicing represents a target for therapy. We describe herein the identification of BPN-15477, a SMC that restores correct splicing of ELP1 exon 20. Using transcriptome sequencing from treated fibroblast cells and a machine learning approach, we identify BPN-15477 responsive sequence signatures. We then leverage this model to discover 155 human disease genes harboring ClinVar mutations predicted to alter pre-mRNA splicing as targets for BPN-15477. Splicing assays confirm successful correction of splicing defects caused by mutations in CFTR, LIPA, MLH1 and MAPT. Subsequent validations in two disease-relevant cellular models demonstrate that BPN-15477 increases functional protein, confirming the clinical potential of our predictions.