Developmental medicine and child neurology · 2026
Passive shoulder mobility and active shoulder external rotation recovery in upper-trunk brachial plexus birth injuries after nerve repair.
СтатьяНазвание и аннотация: Перевод в очереди
Обработка статьи · этапы, ошибки и управление
Загрузка состояния…
Ошибки и ограничения
История · было → стало
Состояние обновляется каждые 5 секунд, пока блок открыт. Процент показывается только там, где известен объём работы. Пауза останавливает продолжение этапа; текущий запрос к источнику может завершиться.
Ulmann Eva T E, Nagels Jochem, Pondaag Willem, Malessy Martijn J A
Аннотация
AIM: To examine the relationship between active shoulder external rotation recovery and passive glenohumeral mobility in children with upper-trunk brachial plexus birth injury (BPBI) after nerve surgery.
METHOD: This retrospective analysis included children with upper-trunk BPBI who underwent primary surgery of the C5 and/or C6 spinal nerves with 2 or more years of follow-up. Linear mixed models were used to analyse passive and active external rotation in adduction at ages 1, 3, 5, 7, and 15 years, adjusted for diagnosis and suprascapular nerve reconstruction strategy (grafting, transfer, neurolysis).
RESULTS: We included 322 children (142 males and 180 females), with a mean (SD) follow-up of 7 years 7 months (4 years 7 months); median age at surgery was 5 months (interquartile range = 4-6 months). Active external rotation increased on average by 2.1° per year (95% confidence interval [CI] = 1.4° to 2.8°, p < 0.001), while passive external rotation decreased on average by 2.6° per year (95% CI = 2.2° to 3.0°, p < 0.001). Passive external rotation was on average 65° (95% CI = 62° to 68°, p < 0.001) greater than active external rotation. The difference decreased by 4.5° per year (95% CI = 3.8° to 5.2°, p < 0.001). At age 15 years, a difference of 36° (95% CI = 29° to 43°) remained.
INTERPRETATION: Passive external rotation is not the explanatory limiting factor in the restoration of active external rotation after nerve surgery in children with upper-trunk BPBI.
METHOD: This retrospective analysis included children with upper-trunk BPBI who underwent primary surgery of the C5 and/or C6 spinal nerves with 2 or more years of follow-up. Linear mixed models were used to analyse passive and active external rotation in adduction at ages 1, 3, 5, 7, and 15 years, adjusted for diagnosis and suprascapular nerve reconstruction strategy (grafting, transfer, neurolysis).
RESULTS: We included 322 children (142 males and 180 females), with a mean (SD) follow-up of 7 years 7 months (4 years 7 months); median age at surgery was 5 months (interquartile range = 4-6 months). Active external rotation increased on average by 2.1° per year (95% confidence interval [CI] = 1.4° to 2.8°, p < 0.001), while passive external rotation decreased on average by 2.6° per year (95% CI = 2.2° to 3.0°, p < 0.001). Passive external rotation was on average 65° (95% CI = 62° to 68°, p < 0.001) greater than active external rotation. The difference decreased by 4.5° per year (95% CI = 3.8° to 5.2°, p < 0.001). At age 15 years, a difference of 36° (95% CI = 29° to 43°) remained.
INTERPRETATION: Passive external rotation is not the explanatory limiting factor in the restoration of active external rotation after nerve surgery in children with upper-trunk BPBI.