JACC. Case reports · 2026
Syncope in an Athlete With Apical Hypertrophic Cardiomyopathy: Ominous or Orthostatic?
Клинический случайСтатьяНазвание и аннотация: Перевод в очереди
Обработка статьи · этапы, ошибки и управление
Загрузка состояния…
Ошибки и ограничения
История · было → стало
Состояние обновляется каждые 5 секунд, пока блок открыт. Процент показывается только там, где известен объём работы. Пауза останавливает продолжение этапа; текущий запрос к источнику может завершиться.
Tatapudi Suhas, Dean John-Henry L, Pabba Krishna, Martinez Matthew W
Аннотация
BACKGROUND: Syncope in athletes with hypertrophic cardiomyopathy (HCM) requires careful evaluation, as etiologies range from benign to life-threatening. Guidelines emphasize individualized risk stratification and shared decision-making.
CASE SUMMARY: A 20-year-old basketball player experienced a syncopal episode when rising from a seated position, preceded by progressive lightheadedness and visual dimming, without chest discomfort or palpitations. Electrocardiogram abnormalities prompted further imaging, confirming the diagnosis of apical HCM. Evaluation demonstrated no left ventricular outflow tract obstruction at rest or with maximal-effort stress testing, and no exertional arrhythmias were observed. The episode was attributed to orthostatic syncope in a preload-dependent structurally abnormal left ventricle. He continued sports participation with hydration counseling, annual imaging, rhythm monitoring, and genetic evaluation, without recurrent events.
DISCUSSION: This case highlights the importance of accurately identifying the etiology of syncope in athletes with HCM and supports individualized, risk informed sport participation rather than universal restriction.
TAKE-HOME MESSAGE: Syncope in athletes with HCM warrants comprehensive evaluation and tailored return-to-play decisions.
CASE SUMMARY: A 20-year-old basketball player experienced a syncopal episode when rising from a seated position, preceded by progressive lightheadedness and visual dimming, without chest discomfort or palpitations. Electrocardiogram abnormalities prompted further imaging, confirming the diagnosis of apical HCM. Evaluation demonstrated no left ventricular outflow tract obstruction at rest or with maximal-effort stress testing, and no exertional arrhythmias were observed. The episode was attributed to orthostatic syncope in a preload-dependent structurally abnormal left ventricle. He continued sports participation with hydration counseling, annual imaging, rhythm monitoring, and genetic evaluation, without recurrent events.
DISCUSSION: This case highlights the importance of accurately identifying the etiology of syncope in athletes with HCM and supports individualized, risk informed sport participation rather than universal restriction.
TAKE-HOME MESSAGE: Syncope in athletes with HCM warrants comprehensive evaluation and tailored return-to-play decisions.