Biomaterials · 2025
Stromal cell-derived factor-encapsulated nanoparticles target ischemic myocardium and attenuate myocardial injury via proangiogenic effects.
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Yajima Shin, Lee Seung Hyun, Yang Junkai, Vergel Matthew D, Manna Manoj K, Kusadokoro Sho, Zhu Yuanjia, Elde Stefan, Mullis Danielle M, Venkatesh Akshay, Ethiraj Sidarth, Ueyama Tsuyoshi, Takashima Hiroyuki, Oh Samuel E, Huynh Chris, Wang Hanjay, Shudo Yasuhiro, Miyagawa Shigeru, Sawa Yoshiki, Rajadas Jayakumar, Woo Y Joseph
Аннотация
Lipid bilayer nanoparticles (NPs) with and without stromal cell-derived factor (SDF) were created to target and treat ischemia/reperfusion (I/R)-injured myocardium. Male Wistar rats were subjected to myocardial I/R insult and, at reperfusion, randomized to receive systemic injections of 5 mL/kg PBS, 6 μg/kg of NPs, SDF, or SDF-NPs. Four days after treatment, SDF-NPs circulated and accumulated selectively in the ischemic myocardium, with an SDF concentration roughly three times that of the other three treatments. SDF-NP-treated rats had more endothelial progenitor cells (EPCs) in the blood and preserved capillaries and arterioles in the ischemic border myocardium four weeks post-treatment, which improved microvascular perfusion, reduced fibrosis, and preserved heart function. Notably, hearts treated with SDF-NPs retained left ventricular function at four weeks compared to 1-day post-treatment, with a 2.7 ± 1.2 % increase in the ejection fraction. The other three treatments decreased left ventricular function at four weeks (PBS: -7.8 ± 1.2 %, P < 0.001; empty NPs: -3.9 ± 1.3 %, P = 0.004; SDF solution: -5.1 ± 1.3 %, P = 0.001). Hence, systemically injected SDF-NPs selectively accumulate in ischemic cardiac tissue, shielding the myocardium from I/R injury via angiogenic effects through increased EPC migration. This novel cardioprotective drug may be clinically translatable.