The Journal of experimental medicine · 2008
Pulmonary alveolar proteinosis caused by deletion of the GM-CSFRalpha gene in the X chromosome pseudoautosomal region 1.
СтатьяПоддержка NIH: внешняя программаНазвание и аннотация: Перевод в очереди
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Martinez-Moczygemba Margarita, Doan Minh L, Elidemir Okan, Fan Leland L, Cheung Sau Wai, Lei Jonathan T, Moore James P, Tavana Ghamartaj, Lewis Lora R, Zhu Yiming, Muzny Donna M, Gibbs Richard A, Huston David P
Аннотация
Pulmonary alveolar proteinosis (PAP) is a rare lung disorder in which surfactant-derived lipoproteins accumulate excessively within pulmonary alveoli, causing severe respiratory distress. The importance of granulocyte/macrophage colony-stimulating factor (GM-CSF) in the pathogenesis of PAP has been confirmed in humans and mice, wherein GM-CSF signaling is required for pulmonary alveolar macrophage catabolism of surfactant. PAP is caused by disruption of GM-CSF signaling in these cells, and is usually caused by neutralizing autoantibodies to GM-CSF or is secondary to other underlying diseases. Rarely, genetic defects in surfactant proteins or the common beta chain for the GM-CSF receptor (GM-CSFR) are causal. Using a combination of cellular, molecular, and genomic approaches, we provide the first evidence that PAP can result from a genetic deficiency of the GM-CSFR alpha chain, encoded in the X-chromosome pseudoautosomal region 1.
Предметные рубрики MeSH
AnimalsCD11b AntigenChild, PreschoolChromosomes, Human, XExonsFemaleGenotypeGranulocyte-Macrophage Colony-Stimulating FactorHumansIn Situ Hybridization, FluorescenceKaryotypingMaleMiceMonocytesPulmonary Alveolar ProteinosisPulmonary SurfactantsReceptors, Granulocyte-Macrophage Colony-Stimulating FactorSignal TransductionTurner Syndrome