European journal of medicinal chemistry · 2026
New chalcogen-optimized 1,3,5-triazines as dual 5-HT6R/FAAH modulators: A versatile approach to neurodegenerative disorders.
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Czarnota-Łydka Kinga, Kucwaj-Brysz Katarzyna, Cios Agnieszka, Mordyl Barbara, Głuch-Lutwin Monika, Jastrzębska-Więsek Magdalena, Partyka Anna, Honkisz-Orzechowska Ewelina, Karcz Tadeusz, Pakulska Julia, Satała Grzegorz, Żesławska Ewa, Dąbrowska Monika, Starek Małgorzata, Więcek Małgorzata, Kurowska Kinga, Pyka Patryk, Brunetti Leonardo, Leuci Rosalba, Piemontese Luca, Nitek Wojciech, Wesołowska Anna, Carrieri Antonio, Handzlik Jadwiga
Аннотация
The clinical failure of selective serotonin 5-HT6 receptor (5-HT6R) antagonists in Alzheimer's disease (AD) highlights the need for multitarget therapeutic strategies addressing the multifactorial nature of neurodegeneration. Building upon our pioneering discovery of the first-in-class dual 5-HT6R/FAAH (fatty acid amide hydrolase) modulators among O-ether triazine compounds, we here report a comprehensive lead-optimization campaign centered on the triazine-based compound MR3b, identified as a promising lead in the search for AD treatment. Structural modifications based on scaffold contraction and chalcogen bioisosterism generated a focused library of 1,3,5-triazine derivatives with diversified GPCR and FAAH profiles. Several compounds displayed nanomolar affinities for 5-HT6R, 5-HT2AR, and D2R, alongside improved FAAH inhibition and antioxidant properties. Sulfur and selenium substitutions markedly enhanced receptor affinity and reduced cytotoxicity compared to the oxygen-containing lead. Selected compounds demonstrated significant neuroprotective effects in cellular models of AD-related pathology, including mitochondrial dysfunction, amyloid-β, and glutamate-induced toxicity. Furthermore, the thio-analogue 4c effectively reversed memory deficits in vivo, showing superior CNS penetration (Kp,brain = 0.78), an expanded therapeutic window (NOR test), and improved safety relative to MR3b. This study identified compound 4c as a second-generation lead and underscores the potential of multitarget triazine-based ligands combining serotonergic modulation and FAAH inhibition as potential disease-modifying candidates for AD and related neurodegenerative disorders.