International immunopharmacology · 2020
Bixin attenuates carbon tetrachloride induced oxidative stress, inflammation and fibrosis in kidney by regulating the Nrf2/TLR4/MyD88 and PPAR-γ/TGF-β1/Smad3 pathway.
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Ma Jie-Qiong, Zhang Yu-Jia, Tian Zhi-Kai, Liu Chan-Min
Аннотация
Bixin, an natural carotenoid extracted from the seeds of the Bixa orellana has been shown to possess numerous important pharmacological activities. The present study was aimed to investigate the mechanisms of Bixin on carbon tetrachloride (CCl4)-induced kidney inflammation, fibrosis and oxidative stress in mice. Our results showed that Bixin improved renal damage by decreasing the serum levels of creatinine, urea, uric acid and alleviating kidney fibrosis. Bixin ameliorated CCl4-induced inflammation in kidneys by reducing the levels of TNF-α and IL-1β. Bixin suppressed oxidative stress by decreasing the MDA level and increasing the activation of SOD, CAT and GPx. Furthermore, Bixin increased the levels of PPAR-γ, NQO1, HO-1 and the nuclear translocation of Nrf2 in the kidneys of mice. Bixin supplementation inhibited the activation of TLR4, MyD88, NF-κB, TGF-β and Smad3. Thus, this study demonstrated that Bixin possesses anti-oxidant, anti-inflammatory and anti-fibrosis properties through regulating the Nrf2/TLR4/MyD88 and PPAR-γ/TGF-β1/Smad3 pathways.
Предметные рубрики MeSH
AnimalsAnti-Inflammatory AgentsAntioxidantsCarbon TetrachlorideCarotenoidsDisease Models, AnimalFibrosisInflammation MediatorsKidneyKidney DiseasesMaleMice, Inbred ICRMyeloid Differentiation Factor 88NF-E2-Related Factor 2Oxidative StressPPAR gammaSignal TransductionSmad3 ProteinToll-Like Receptor 4Transforming Growth Factor beta1Mice