Leukemia · 2015
Molecular profiling of myeloid progenitor cells in multi-mutated advanced systemic mastocytosis identifies KIT D816V as a distinct and late event.
СтатьяПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
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Jawhar M, Schwaab J, Schnittger S, Sotlar K, Horny H-P, Metzgeroth G, Müller N, Schneider S, Naumann N, Walz C, Haferlach T, Valent P, Hofmann W-K, Cross N C P, Fabarius A, Reiter A
Аннотация
To explore the molecular profile and its prognostic implication in systemic mastocytosis (SM), we analyzed the mutation status of granulocyte-macrophage colony-forming progenitor cells (CFU-GM) in patients with KIT D816V(+) indolent SM (ISM, n=4), smoldering SM (SSM, n=2), aggressive SM (ASM, n=1), SM with associated clonal hematologic non-mast cell lineage disorder (SM-AHNMD, n=5) and ASM-AHNMD (n=7). All patients with (A)SM-AHNMD (n=12) carried 1-4 (median 3) additional mutations in 11 genes tested, most frequently TET2, SRSF2, ASXL1, CBL and EZH2. In multi-mutated (A)SM-AHNMD, KIT D816V(+) single-cell-derived CFU-GM colonies were identified in 8/12 patients (median 60%, range 0-95). Additional mutations were identified in CFU-GM colonies in all patients, and logical hierarchy analysis indicated that mutations in TET2, SRSF2 and ASXL1 preceded KIT D816V. In ISM/SSM, no additional mutations were detected and CFU-GM colonies were exclusively KIT D816V(-). These data indicate that (a) (A)SM-AHNMD is a multi-mutated neoplasm, (b) mutations in TET2, SRSF2 or ASXL1 precede KIT D816V in ASM-AHNMD,
Предметные рубрики MeSH
AgedCell LineageCell SeparationCytogeneticsDNA Mutational AnalysisDNA-Binding ProteinsDioxygenasesFemaleFlow CytometryGenotypeGranulocyte-Macrophage Progenitor CellsHumansIn Situ Hybridization, FluorescenceMaleMast CellsMastocytosis, SystemicMiddle AgedMutationNuclear ProteinsPhenotypePrognosisProto-Oncogene ProteinsProto-Oncogene Proteins c-kitRepressor ProteinsRibonucleoproteinsSerine-Arginine Splicing FactorsStem Cells