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DOI 10.1155/2022/6499668

Медицинская библиотека · Mediators of inflammation · 2022

The Role of Cytokines in Nephrotic Syndrome.

Ahmadian Elham, Rahbar Saadat Yalda, Dalir Abdolahinia Elaheh, Bastami Milad, Shoja Mohammadali M, Zununi Vahed Sepideh, Ardalan Mohammadreza

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Аннотация

Idiopathic nephrotic syndrome (INS) is an important primary glomerular disease characterized by severe proteinuria. Evidence supports a role for T cell dysfunction in the pathogenesis of INS. Glucocorticoids are the primary therapy for INS; however, steroid-resistant NS (SRNS) patients are at a higher risk of drug-induced side effects and harbor poor prognosis. Although the exact mechanism of the resistance is unknown, the imbalances of T helper subtype 1 (Th1), Th2, and regulatory T cells (Tregs) and their cytokines may be involved in the pathogenesis of glucocorticoid responsiveness. Up to now, no confirmed biomarkers have been able to predict SRNS; however, a panel of cytokines may predict responsiveness and identify SRNS patients. Thus, the introduction of distinctive cytokines as novel biomarkers of SRNS enables both preventions of drug-related toxicity and earlier switch to more effective therapies. This review highlights the impacts of T cell population imbalances and their downstream cytokines on response to glucocorticoid responsiveness state in INS.

Для цитирования: Ahmadian Elham, Rahbar Saadat Yalda, Dalir Abdolahinia Elaheh, Bastami Milad, Shoja Mohammadali M, Zununi Vahed Sepideh, Ardalan Mohammadreza. The Role of Cytokines in Nephrotic Syndrome.. Mediators of inflammation. 2022. DOI: 10.1155/2022/6499668.

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Обработка статьи · этапы, ошибки и управление
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