Voiding dysfunction: patients with human T-lymphotropic-virus-type-1-associated myelopathy.

Voiding dysfunction was evaluated in 26 patients (9 males and 17 females) with human T-lymphotropic-virus-type-1-associated myelopathy. Of 26 patients 22 (85%) had difficulty in urination, 15 (58%) had urinary frequency and 9 (35%) had urge incontinence. Cystograms showed trabeculated bladder in 5 patients, vesicoureteral reflux in 3 and bladder neck obstruction in 5. In 25 patients (96%) urodynamic studies showed detrusor hyperreflexia with normal urethral function during storage. Of these patients 17 had detrusor underactivity with detrusor-sphincter dyssynergia during micturition. One patient had normal detrusor function during storage and detrusor areflexia during voiding.