Biochemical and biophysical research communications · 2014
Polyomavirus BK-encoded microRNA suppresses autoregulation of viral replication.
СтатьяПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
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Tian Ya-Chung, Li Yi-Jung, Chen Hua-Chien, Wu Hsin-Hsu, Weng Cheng-Hao, Chen Yung-Chang, Lee Cheng-Chia, Chang Ming-Yang, Hsu Hsiang-Hao, Yen Tzung-Hai, Hung Cheng-Chieh, Yang Chih-Wei
Аннотация
Polyomavirus BK (BKV) infection is an important cause of renal allograft failure. Viral microRNAs are known to play a crucial role in viral replication. This study investigated the expression of BKV-encoded microRNAs (miR-B1) in patients with polyomavirus-associated nephropathy (PVAN) and their role in viral replication. Following BKV infection in renal proximal tubular cells, the 3p and 5p miR-B1 levels were significantly increased. Cells transfected with the vector containing the miR-B1 precursor (the miR-B1 vector) showed a significant increase in expression of 3p and 5p miR-B1 and decrease in luciferase activity of a reporter containing the 3p and 5p miR-B1 binding sites, compared to cells transfected with the miR-B1-mutated vector. Transfection of the miR-B1 expression vector or the 3p and 5p miR-B1 oligonucleotides inhibited expression of TAg. TAg-enhanced promoter activity and BKV replication were inhibited by miR-B1. In contrast, inhibition of miR-B1 expression by addition of miR-B1 antagomirs or silencing of Dicer upregulated the expression of TAg and VP1 proteins in BKV-infected cells. Importantly, patients with PVAN had significantly higher levels of 3p and 5p miR-B1 compared to renal transplant patients without PVAN. In conclusion, we demonstrated that (1) miR-B1 expression was upregulated during BKV infection and (2) miR-B1 suppressed TAg-mediated autoregulation of BKV replication. Use of miR-B1 can be evaluated as a potential treatment strategy against BKV infection.
Предметные рубрики MeSH
Antigens, Viral, TumorBK VirusCapsid ProteinsDEAD-box RNA HelicasesDown-RegulationGene Expression Regulation, ViralGene SilencingGraft RejectionHomeostasisHumansKidney TransplantationMicroRNAsPolyomavirus InfectionsProtein BiosynthesisRNA, ViralRibonuclease IIITranscription, GeneticVirus Replication