Diabetes · 2015
Apelin Controls Fetal and Neonatal Glucose Homeostasis and Is Altered by Maternal Undernutrition.
СтатьяПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
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Mayeur Sylvain, Wattez Jean-Sébastien, Lukaszewski Marie-Amélie, Lecoutre Simon, Butruille Laura, Drougard Anne, Eberlé Delphine, Bastide Bruno, Laborie Christine, Storme Laurent, Knauf Claude, Vieau Didier, Breton Christophe, Lesage Jean
Аннотация
The adequate control of glucose homeostasis during both gestation and early postnatal life is crucial for the development of the fetoplacental unit and adaptive physiological responses at birth. Growing evidences indicate that apelin and its receptor, APJ, which are expressed across a wide range of tissues, exert important roles in glucose homeostasis in adults. However, little is known about the function of the apelinergic system during gestation. In this study, we evaluated the activity of this system in rats, the role of apelin in fetal and neonatal glucose homeostasis, and its modulation by maternal food restriction. We found that 1) the apelinergic system was expressed at the fetoplacental interface and in numerous fetal tissues, 2) ex vivo, the placenta released high amounts of apelin in late gestation, 3) intravenous apelin injection in mothers increased the transplacental transport of glucose, and 4) intraperitoneal apelin administration in neonates increased glucose uptake in lung and muscle. Maternal food restriction drastically reduced apelinemia in both mothers and growth-restricted fetuses and altered the expression of the apelinergic system at the fetoplacental interface. Together, our data demonstrate that apelin controls fetal and neonatal glucose homeostasis and is altered by fetal growth restriction induced by maternal undernutrition.
Предметные рубрики MeSH
AnimalsAnimals, NewbornApelinApelin ReceptorsBlood GlucoseEnzyme-Linked Immunosorbent AssayFemaleFetal Growth RetardationFetusGene Expression Regulation, DevelopmentalGlucose Transporter Type 1Glucose Transporter Type 3HomeostasisInsulinIntercellular Signaling Peptides and ProteinsLungMalnutritionMuscle, SkeletalPlacentaPregnancyPregnancy ComplicationsRatsRats, WistarReceptors, G-Protein-CoupledReverse Transcriptase Polymerase Chain Reaction