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DOI 10.1155/2020/4957238

Медицинская библиотека · Oxidative medicine and cellular longevity · 2020

Diazoxide Protects against Myocardial Ischemia/Reperfusion Injury by Moderating ERS via Regulation of the miR-10a/IRE1 Pathway.

Zhang Lin, Cai Shuang, Cao Song, Nie Jia, Zhou Wenjing, Zhang Yu, Li Ke, Wang Haiying, Yu Shouyang, Yu Tian

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Аннотация

Nowadays, reperfusion is still the most effective treatment for ischemic heart disease. However, cardiac reperfusion therapy would lead to reperfusion injury, which may have resulted from endoplasmic reticulum stress (ERS) during reperfusion. Diazoxide (DZ) is a highly selective mitochondrial adenosine triphosphate-sensitive potassium channel opener. Its protective effect on I/R injury has been confirmed in many organs such as the heart and brain. However, the mechanism of its protective effect has not been fully elucidated. MicroRNAs (miRNAs) are widely involved in pathologies of heart disease. In this study, we found that miR-10a expression was highly upregulated in the myocardial I/R groups, and DZ treatment significantly reduced the expression of miR-10a. More importantly, we found that DZ treatment can moderate ERS via regulation of the miR-10a/IRE1 pathway in the I/R and H/R models, thereby protecting myocardial H/R injury.

Для цитирования: Zhang Lin, Cai Shuang, Cao Song, Nie Jia, Zhou Wenjing, Zhang Yu, Li Ke, Wang Haiying, Yu Shouyang, Yu Tian. Diazoxide Protects against Myocardial Ischemia/Reperfusion Injury by Moderating ERS via Regulation of the miR-10a/IRE1 Pathway.. Oxidative medicine and cellular longevity. 2020. DOI: 10.1155/2020/4957238.

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