The Journal of clinical investigation · 2020
Cholinergic dysfunction in the dorsal striatum promotes habit formation and maladaptive eating.
СтатьяПоддержка NIH: внешняя программаПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
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Favier Mathieu, Janickova Helena, Justo Damian, Kljakic Ornela, Runtz Léonie, Natsheh Joman Y, Pascoal Tharick A, Germann Jurgen, Gallino Daniel, Kang Jun-Ii, Meng Xiang Qi, Antinora Christina, Raulic Sanda, Jacobsen Jacob Pr, Moquin Luc, Vigneault Erika, Gratton Alain, Caron Marc G, Duriez Philibert, Brandon Mark P, Neto Pedro Rosa, Chakravarty M Mallar, Herzallah Mohammad M, Gorwood Philip, Prado Marco Am, Prado Vania F, El Mestikawy Salah
Аннотация
Dysregulation of habit formation has been recently proposed as pivotal to eating disorders. Here, we report that a subset of patients suffering from restrictive anorexia nervosa have enhanced habit formation compared with healthy controls. Habit formation is modulated by striatal cholinergic interneurons. These interneurons express vesicular transporters for acetylcholine (VAChT) and glutamate (VGLUT3) and use acetylcholine/glutamate cotransmission to regulate striatal functions. Using mice with genetically silenced VAChT (VAChT conditional KO, VAChTcKO) or VGLUT3 (VGLUT3cKO), we investigated the roles that acetylcholine and glutamate released by cholinergic interneurons play in habit formation and maladaptive eating. Silencing glutamate favored goal-directed behaviors and had no impact on eating behavior. In contrast, VAChTcKO mice were more prone to habits and maladaptive eating. Specific deletion of VAChT in the dorsomedial striatum of adult mice was sufficient to phenocopy maladaptive eating behaviors of VAChTcKO mice. Interestingly, VAChTcKO mice had reduced dopamine release in the dorsomedial striatum but not in the dorsolateral striatum. The dysfunctional eating behavior of VAChTcKO mice was alleviated by donepezil and by l-DOPA, confirming an acetylcholine/dopamine deficit. Our study reveals that loss of acetylcholine leads to a dopamine imbalance in striatal compartments, thereby promoting habits and vulnerability to maladaptive eating in mice.