International journal of cardiology · 2007
Cardiac rhabdomyoma in tuberous sclerosis: hyperactive Erk signaling.
Клинический случайПисьмо в редакциюПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
Обработка статьи · этапы, ошибки и управление
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Jozwiak Jaroslaw, Sahin Mustafa, Jozwiak Sergiusz, Kotulska Katarzyna, Ploski Rafal, Szperl Malgorzata, Wlodarski Pawel K
Аннотация
Tuberous sclerosis (TS) is a neurological disorder associated with the formation of tumors in several organs. Cardiac rhabdomyomas are possibly the earliest symptom of TS. Although rhabdomyomas are present in about half of TS patients, little is known of their molecular background since these tumors are rarely resected. Here we present a patient diagnosed with TS, in whom rhabdomyoma has been excised due to deterioration of hemodynamics. We found, that the tumor remained heterozygous for the affected TSC2 gene. To analyze molecular mechanisms implicated in rhabdomyoma growth, we determined the status of mTOR, Akt and Erk pathways. We found that Akt was not upregulated, while mTOR, Erk and its substrates were hyperactive. Classic activator of Erk, MEK, was only modestly active. We hypothesize that rhabdomyoma arising in TS may progress due to Erk potentiation.
Предметные рубрики MeSH
Adaptor Proteins, Signal TransducingApoptosis Regulatory ProteinsChild, PreschoolExtracellular Signal-Regulated MAP KinasesFemaleHeart NeoplasmsHumansIntracellular Signaling Peptides and ProteinsProtein Serine-Threonine KinasesProto-Oncogene Proteins c-aktRhabdomyomaSignal TransductionTOR Serine-Threonine KinasesTuberous SclerosisTuberous Sclerosis Complex 2 ProteinTumor Suppressor Proteins