Frontiers in cellular and infection microbiology · 2018
MptpB Promotes Mycobacteria Survival by Inhibiting the Expression of Inflammatory Mediators and Cell Apoptosis in Macrophages.
СтатьяПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
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Fan Lingbo, Wu Xiaoyu, Jin Chunyan, Li Fengge, Xiong Sidong, Dong Yuanshu
Аннотация
Tuberculosis is a severe contagious disease caused by Mycobacterium tuberculosis (Mtb). To develop new vaccines and medicine against TB, there is an urgent need to provide insights into the mechanisms by which Mtb induces tuberculosis. In this study, we found that secreted Mtb virulence factor MptpB significantly enhanced the survival of H37Rv in macrophages. MptpB suppressed the production of iNOS, the expression of inflammatory factors IL-1β and IL-6, as well as the apoptosis of the macrophage in Mtb infected RAW264.7 cells. Mechanism investigation showed that MptpB simultaneously hampered the NF-κB and MAPK signal pathways, evidenced by its blocking of p65, IKKα, Erk1/2, and p38 phosphorylation induced by Mtb infection. MptpB also inhibited host cell p53 expression. The results demonstrated that MptpB contributed to the survival of H37Rv by inhibiting host inflammatory responses and apoptosis through impeding the NF-κB and MAPK signal pathways and p53 expression in the macrophage.
Предметные рубрики MeSH
AnimalsApoptosisBacterial ProteinsCytokinesI-kappa B KinaseInflammationInflammation MediatorsInterleukin-1betaInterleukin-6MAP Kinase Signaling SystemMacrophage ActivationMacrophagesMiceMitogen-Activated Protein KinasesMycobacterium smegmatisMycobacterium tuberculosisNF-kappa BNitric Oxide Synthase Type IIProtein Tyrosine PhosphatasesRAW 264.7 CellsSignal TransductionTuberculosisVirulence Factors