Anti-cancer drugs · 2009
Gene expression-signature of belinostat in cell lines is specific for histone deacetylase inhibitor treatment, with a corresponding signature in xenografts.
СтатьяПоддержка NIH: внешняя программаНазвание и аннотация: Перевод в очереди
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Monks Anne, Hose Curtis D, Pezzoli Patrick, Kondapaka Sudhir, Vansant Gordon, Petersen Kamille Dumong, Sehested Maxwell, Monforte Joseph, Shoemaker Robert H
Аннотация
Belinostat is a hydroxamate-type histone deactylase inhibitor (HDACi), which has recently entered phase I and II clinical trials. Microarray-based analysis of belinostat-treated cell lines showed an impact on genes associated with the G2/M phase of the cell cycle and downregulation of the aurora kinase pathway. Expression of 25 dysregulated genes was measured in eight differentially sensitive cell lines using a novel high-throughput assay that combines multiplex reverse transcriptase-PCR and fluorescence capillary electrophoresis. Sensitivity to belinostat and the magnitude of changes in overall gene modulation were significantly correlated. A belinostat-gene profile was specific for HDACi in three cell lines when compared with equipotent concentrations of four mechanistically different chemotherapeutic agents: 5-fluorouracil, cisplatin, paclitaxel, and thiotepa. Belinostat- and trichostatin A (HDACi)-induced gene responses were highly correlated with each other, but not with the limited changes in response to the other non-HDACi agents. Moreover, belinostat treatment of mice bearing human xenografts showed that the preponderance of selected genes were also modulated in vivo, more extensively in a drug-sensitive tumor than a more resistant model. We have demonstrated a gene signature that is selectively regulated by HDACi when compared with other clinical agents allowing us to distinguish HDACi responses from those related to other mechanisms.
Предметные рубрики MeSH
AnimalsAntineoplastic AgentsAurora KinasesCell CycleCell Cycle ProteinsCell Line, TumorCell ProliferationChromosomal Proteins, Non-HistoneDown-RegulationEnzyme InhibitorsFemaleGene ExpressionGene Expression ProfilingGuanine Nucleotide Exchange FactorsHCT116 CellsHistone Deacetylase InhibitorsHumansHydroxamic AcidsMaleMiceMice, Inbred StrainsNeoplasmsProtein Serine-Threonine KinasesReverse Transcriptase Polymerase Chain ReactionSulfonamidesUp-RegulationXenograft Model Antitumor Assaysran GTP-Binding Protein