The EMBO journal · 2026
Hox/Meis-dependent gene-regulatory transition underlies cardiopharyngeal neural crest diversification.
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Iwase Akiyasu, Uchijima Yasunobu, Seya Daiki, Kida Mayuko, Higashiyama Hiroki, Matsui Kazuhiro, Taguchi Akashi, Harada Yukihiro, Wang Yunce, Yamamoto Shogo, Fukuda Shiro, Nomura Seitaro, Kohro Takahide, Shukunami Chisa, Akiyama Haruhiko, Seki Masahide, Kanai Akinori, Suzuki Yutaka, Kawamura Teruhisa, Nakagawa Osamu, Katoh Hiroto, Ishikawa Shumpei, Wada Youichiro, Aburatani Hiroyuki, Kurihara Yukiko, Miyagawa-Tomita Sachiko, Kurihara Hiroki
Аннотация
Neural crest cells (NCCs) are multipotent migratory cells essential for cardiac development, yet the lineage trajectories and gene regulatory networks underlying their differentiation in the cardiopharyngeal region remain unclear. Here, we integrate single-cell RNA-seq, spatial transcriptomics, and multiomic analyses to construct a comprehensive map of NCC lineages in developing mouse cardiopharyngeal tissues. We identify a transition from Hox-positive pharyngeal NCCs to Hox-negative intracardiac populations associated with the outflow tract cushion, accompanied by a shift in Meis transcription factor binding and gene-regulatory network architecture. By contrast, NCCs forming the aorticopulmonary septum and great vessel smooth muscle retain distinct Hox-codes. A Meis2-Sox9-Scx gene-regulatory network defines a skeletogenic progenitor-like intermediate state that gives rise to coronary artery smooth muscle and semilunar valves. Our findings suggest that the loss of Hox-dependent regional identity enables pharyngeal NCCs to acquire new fates upon entering the cardiac cushion, providing insight into the developmental origins of coronary and valvular calcification.