Otology & neurotology : official publication of the American Otological Society, American Neurotology Society [and] European Academy of Otology and Neurotology · 2001
An anatomic variant of the anterior inferior cerebellar artery in a patient with Ménière's disease.
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Scoleri P, Widner S A, Cass S P
Аннотация
OBJECTIVE: To describe an anatomic variant of the anterior inferior cerebellar artery in a patient with Ménière's disease.
STUDY DESIGN: Retrospective case review and review of the literature.
SETTING: Tertiary referral clinic.
INTERVENTION: Vestibular nerve section and microvascular decompression.
MAIN OUTCOME MEASURES: Audiometric testing and control of vertigo.
RESULTS: The eighth nerve was identified via a retromastoid approach. The anterior inferior cerebellar artery was observed bisecting the eighth nerve. The vestibular nerve was sectioned, and microvascular decompression was performed on the cochlear division. At last follow-up, the patient had not experienced any vertiginous attacks but was observed to have progressive hearing loss.
CONCLUSIONS: The course of the anterior inferior cerebellar artery is highly variable and difficult to predict. Knowing the potential paths is a necessity in performing posterior fossa surgery. Although the patient's vertigo was controlled by the vestibular nerve section, microvascular decompression of the cochlear nerve did not result in hearing improvement or stabilization. This case report does not support a benefit of microvascular decompression in Méniére's disease. Vestibular nerve section remains the authors' treatment of choice for controlling disabling vertigo caused by Ménière's disease.
STUDY DESIGN: Retrospective case review and review of the literature.
SETTING: Tertiary referral clinic.
INTERVENTION: Vestibular nerve section and microvascular decompression.
MAIN OUTCOME MEASURES: Audiometric testing and control of vertigo.
RESULTS: The eighth nerve was identified via a retromastoid approach. The anterior inferior cerebellar artery was observed bisecting the eighth nerve. The vestibular nerve was sectioned, and microvascular decompression was performed on the cochlear division. At last follow-up, the patient had not experienced any vertiginous attacks but was observed to have progressive hearing loss.
CONCLUSIONS: The course of the anterior inferior cerebellar artery is highly variable and difficult to predict. Knowing the potential paths is a necessity in performing posterior fossa surgery. Although the patient's vertigo was controlled by the vestibular nerve section, microvascular decompression of the cochlear nerve did not result in hearing improvement or stabilization. This case report does not support a benefit of microvascular decompression in Méniére's disease. Vestibular nerve section remains the authors' treatment of choice for controlling disabling vertigo caused by Ménière's disease.