Brain : a journal of neurology · 2021
The GGC repeat expansion in NOTCH2NLC is associated with oculopharyngodistal myopathy type 3.
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Yu Jiaxi, Deng Jianwen, Guo Xueyu, Shan Jingli, Luan Xinghua, Cao Li, Zhao Juan, Yu Meng, Zhang Wei, Lv He, Xie Zhiying, Meng LingChao, Zheng Yiming, Zhao Yawen, Gang Qiang, Wang Qingqing, Liu Jing, Zhu Min, Zhou Binbin, Li Pidong, Liu Yinzhe, Wang Yang, Yan Chuanzhu, Hong Daojun, Yuan Yun, Wang Zhaoxia
Аннотация
Oculopharyngodistal myopathy (OPDM) is an adult-onset neuromuscular disease characterized by progressive ocular, facial, pharyngeal and distal limb muscle involvement. Trinucleotide repeat expansions in LRP12 or GIPC1 were recently reported to be associated with OPDM. However, a significant portion of OPDM patients have unknown genetic causes. In this study, long-read whole-genome sequencing and repeat-primed PCR were performed and we identified GGC repeat expansions in the NOTCH2NLC gene in 16.7% (4/24) of a cohort of Chinese OPDM patients, designated as OPDM type 3 (OPDM3). Methylation analysis indicated that methylation levels of the NOTCH2NLC gene were unaltered in OPDM3 patients, but increased significantly in asymptomatic carriers. Quantitative real-time PCR analysis indicated that NOTCH2NLC mRNA levels were increased in muscle but not in blood of OPDM3 patients. Immunofluorescence on OPDM muscle samples and expressing mutant NOTCH2NLC with (GGC)69 repeat expansions in HEK293 cells indicated that mutant NOTCH2NLC-polyglycine protein might be a major component of intranuclear inclusions, and contribute to toxicity in cultured cells. In addition, two RNA-binding proteins, hnRNP A/B and MBNL1, were both co-localized with p62 in intranuclear inclusions in OPDM muscle samples. These results indicated that a toxic protein gain-of-function mechanism and RNA gain-of-function mechanism may both play a vital role in the pathogenic processes of OPDM3. This study extended the spectrum of NOTCH2NLC repeat expansion-related diseases to a predominant myopathy phenotype presenting as OPDM, and provided evidence for possible pathogenesis of these diseases.