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Proceedings of the National Academy of Sciences of the United States of America · 2021

A nonsense TMEM43 variant leads to disruption of connexin-linked function and autosomal dominant auditory neuropathy spectrum disorder.

СтатьяПоддержка NIH: внешняя программаПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод пока не подготовлен
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Jang Minwoo Wendy, Oh Doo-Yi, Yi Eunyoung, Liu Xuezhong, Ling Jie, Kim Nayoung, Sharma Kushal, Kim Tai Young, Lee Seungmin, Kim Ah-Reum, Kim Min Young, Kim Min-A, Lee Mingyu, Han Jin-Hee, Han Jae Joon, Park Hye-Rim, Kim Bong Jik, Lee Sang-Yeon, Woo Dong Ho, Oh Jayoung, Oh Soo-Jin, Du Tingting, Koo Ja-Won, Oh Seung-Ha, Shin Hyun-Woo, Seong Moon-Woo, Lee Kyu-Yup, Kim Un-Kyung, Shin Jung Bum, Sang Shushan, Cai Xinzhang, Mei Lingyun, He Chufeng, Blanton Susan H, Chen Zheng-Yi, Chen Hongsheng, Liu Xianlin, Nourbakhsh Aida, Huang Zaohua, Kang Kwon-Woo, Park Woong-Yang, Feng Yong, Lee C Justin, Choi Byung Yoon

Аннотация

Genes that are primarily expressed in cochlear glia-like supporting cells (GLSs) have not been clearly associated with progressive deafness. Herein, we present a deafness locus mapped to chromosome 3p25.1 and an auditory neuropathy spectrum disorder (ANSD) gene, TMEM43, mainly expressed in GLSs. We identify p.(Arg372Ter) of TMEM43 by linkage analysis and exome sequencing in two large Asian families segregating ANSD, which is characterized by inability to discriminate speech despite preserved sensitivity to sound. The knock-in mouse with the p.(Arg372Ter) variant recapitulates a progressive hearing loss with histological abnormalities in GLSs. Mechanistically, TMEM43 interacts with the Connexin26 and Connexin30 gap junction channels, disrupting the passive conductance current in GLSs in a dominant-negative fashion when the p.(Arg372Ter) variant is introduced. Based on these mechanistic insights, cochlear implant was performed on three subjects, and speech discrimination was successfully restored. Our study highlights a pathological role of cochlear GLSs by identifying a deafness gene and its causal relationship with ANSD.

Предметные рубрики MeSH

ИП Урсолов Владлен Сергеевич

ИНН 500118482352 · ОГРНИП 314500117700049

Реквизиты и контакты