Journal of neural transmission (Vienna, Austria : 1996) · 2021
Investigation of gene-environment interactions in relation to tic severity.
СтатьяМетаанализПоддержка исследования: вне правительства СШАПоддержка NIH: внешняя программаНазвание и аннотация: Перевод пока не подготовлен
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Abdulkadir Mohamed, Yu Dongmei, Osiecki Lisa, King Robert A, Fernandez Thomas V, Brown Lawrence W, Cheon Keun-Ah, Coffey Barbara J, Garcia-Delgar Blanca, Gilbert Donald L, Grice Dorothy E, Hagstrøm Julie, Hedderly Tammy, Heyman Isobel, Hong Hyun Ju, Huyser Chaim, Ibanez-Gomez Laura, Kim Young Key, Kim Young-Shin, Koh Yun-Joo, Kook Sodahm, Kuperman Samuel, Leventhal Bennett, Madruga-Garrido Marcos, Maras Athanasios, Mir Pablo, Morer Astrid, Münchau Alexander, Plessen Kerstin J, Roessner Veit, Shin Eun-Young, Song Dong-Ho, Song Jungeun, Visscher Frank, Zinner Samuel H, Mathews Carol A, Scharf Jeremiah M, Tischfield Jay A, Heiman Gary A, Dietrich Andrea, Hoekstra Pieter J
Аннотация
Tourette syndrome (TS) is a neuropsychiatric disorder with involvement of genetic and environmental factors. We investigated genetic loci previously implicated in Tourette syndrome and associated disorders in interaction with pre- and perinatal adversity in relation to tic severity using a case-only (N = 518) design. We assessed 98 single-nucleotide polymorphisms (SNPs) selected from (I) top SNPs from genome-wide association studies (GWASs) of TS; (II) top SNPs from GWASs of obsessive-compulsive disorder (OCD), attention-deficit/hyperactivity disorder (ADHD), and autism spectrum disorder (ASD); (III) SNPs previously implicated in candidate-gene studies of TS; (IV) SNPs previously implicated in OCD or ASD; and (V) tagging SNPs in neurotransmitter-related candidate genes. Linear regression models were used to examine the main effects of the SNPs on tic severity, and the interaction effect of these SNPs with a cumulative pre- and perinatal adversity score. Replication was sought for SNPs that met the threshold of significance (after correcting for multiple testing) in a replication sample (N = 678). One SNP (rs7123010), previously implicated in a TS meta-analysis, was significantly related to higher tic severity. We found a gene-environment interaction for rs6539267, another top TS GWAS SNP. These findings were not independently replicated. Our study highlights the future potential of TS GWAS top hits in gene-environment studies.