Nature communications · 2020
Microbiota-induced tissue signals regulate ILC3-mediated antigen presentation.
СтатьяПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод пока не подготовлен
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Lehmann Frank Michael, von Burg Nicole, Ivanek Robert, Teufel Claudia, Horvath Edit, Peter Annick, Turchinovich Gleb, Staehli Daniel, Eichlisberger Tobias, Gomez de Agüero Mercedes, Coto-Llerena Mairene, Prchal-Murphy Michaela, Sexl Veronika, Bentires-Alj Mohamed, Mueller Christoph, Finke Daniela
Аннотация
Although group 3 innate lymphoid cells (ILC3s) are efficient inducers of T cell responses in the spleen, they fail to induce CD4+ T cell proliferation in the gut. The signals regulating ILC3-T cell responses remain unknown. Here, we show that transcripts associated with MHC II antigen presentation are down-modulated in intestinal natural cytotoxicity receptor (NCR)- ILC3s. Further data implicate microbiota-induced IL-23 as a crucial signal for reversible silencing of MHC II in ILC3s, thereby reducing the capacity of ILC3s to present antigen to T cells in the intestinal mucosa. Moreover, IL-23-mediated MHC II suppression is dependent on mTORC1 and STAT3 phosphorylation in NCR- ILC3s. By contrast, splenic interferon-γ induces MHC II expression and CD4+ T cell stimulation by NCR- ILC3s. Our results thus identify biological circuits for tissue-specific regulation of ILC3-dependent T cell responses. These pathways may have implications for inducing or silencing T cell responses in human diseases.
Предметные рубрики MeSH
AnimalsAntigen PresentationAntigen-Presenting CellsAntigens, CDCell PolarityDown-RegulationHistocompatibility Antigens Class IIImmunity, InnateInterferon-gammaInterleukin-23Lymphocyte ActivationLymphocytesMechanistic Target of Rapamycin Complex 1MiceMicrobiotaNuclear ProteinsPhenotypePhosphorylationPrincipal Component AnalysisPromoter Regions, GeneticRNA, MessengerSTAT3 Transcription FactorSpleenT-LymphocytesTrans-ActivatorsTranscription, Genetic