Molecules (Basel, Switzerland) · 2023
Targeting HIV-1 Reverse Transcriptase Using a Fragment-Based Approach.
СтатьяНазвание и аннотация: Перевод в очереди
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Mansouri Mahta, Rumrill Shawn, Dawson Shane, Johnson Adam, Pinson Jo-Anne, Gunzburg Menachem J, Latham Catherine F, Barlow Nicholas, Mbogo George W, Ellenberg Paula, Headey Stephen J, Sluis-Cremer Nicolas, Tyssen David, Bauman Joseph D, Ruiz Francesc X, Arnold Eddy, Chalmers David K, Tachedjian Gilda
Аннотация
Human immunodeficiency virus type I (HIV-1) is a retrovirus that infects cells of the host's immune system leading to acquired immunodeficiency syndrome and potentially death. Although treatments are available to prevent its progression, HIV-1 remains a major burden on health resources worldwide. Continued emergence of drug-resistance mutations drives the need for novel drugs that can inhibit HIV-1 replication through new pathways. The viral protein reverse transcriptase (RT) plays a fundamental role in the HIV-1 replication cycle, and multiple approved medications target this enzyme. In this study, fragment-based drug discovery was used to optimize a previously identified hit fragment (compound B-1), which bound RT at a novel site. Three series of compounds were synthesized and evaluated for their HIV-1 RT binding and inhibition. These series were designed to investigate different vectors around the initial hit in an attempt to improve inhibitory activity against RT. Our results show that the 4-position of the core scaffold is important for binding of the fragment to RT, and a lead compound with a cyclopropyl substitution was selected and further investigated. Requirements for binding to the NNRTI-binding pocket (NNIBP) and a novel adjacent site were investigated, with lead compound 27-a minimal but efficient NNRTI-offering a starting site for the development of novel dual NNIBP-Adjacent site inhibitors.