Biallelic variants in RNU2-2 cause the most prevalent known recessive neurodevelopmental disorder.
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Greene Daniel, Mendez Rodrigo, Lees Jon, Barbosa Mafalda, Bruselles Alessandro, Chiriatti Luigi, Ferraro Federico, Mancini Cecilia, Schot Rachel, Sleutels Frank, Bertini Enrico, Bonner Devon E, Bouman Arjan, Brooks Alice S, Cassini Thomas A, Ezell Kimberly M, Gomez-Ospina Natalia, Kleefstra Tjitske, O'Donoghue Michael, Rives Lynette, Shashi Vandana, Spillmann Rebecca C, Wafik Mohamed, Undiagnosed Diseases Network, Freson Kathleen, Barakat Tahsin Stefan, Tartaglia Marco, Bernstein Jonathan A, Mumford Andrew D, Wheeler Matthew T, Turro Ernest
Аннотация
We recently showed that mutations in the snRNA genes RNU4-2 and RNU2-2 are prevalent causes of dominant neurodevelopmental disorders (NDDs). Here, by genetic association, we demonstrate the existence of a recessive form of RNU2-2 syndrome. We inferred a log Bayes factor for a recessive model of association of 18.2. Conditional on that model, 17 rare variants had a posterior probability of pathogenicity >0.8. This conservative threshold identified 18 probands and 5 affected siblings, each carrying two alleles in trans at these variants. A relaxed threshold of >0.6 identified a further 13 candidate probands. We identified nine further cases in replication collections. Affected individuals have intellectual disability, global developmental delay and seizures. Recessive RNU2-2 syndrome accounts for ~10% of families with a recessive NDD presently diagnosable by sequencing and affects ~60% as many families as the dominant RNU4-2-related NDD ReNU syndrome. The variants are predicted to destabilize stem loops and binding domains of U2-2 snRNA. Whole-blood RNA sequencing data showed a >90% reduction in the expression of pathogenic U2-2 alleles in biallelic cases and monoallelic carriers, albeit with wild-type compensation in carriers, pointing to a loss-of-expression mechanism.