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Nature structural & molecular biology · 2024

African ancestry neurodegeneration risk variant disrupts an intronic branchpoint in GBA1.

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Álvarez Jerez Pilar, Wild Crea Peter, Ramos Daniel M, Gustavsson Emil K, Radefeldt Mandy, Damianov Andrey, Makarious Mary B, Ojo Oluwadamilola O, Billingsley Kimberley J, Malik Laksh, Daida Kensuke, Bromberek Sarah, Hu Fangle, Schneider Zachary, Surapaneni Aditya L, Stadler Julia, Rizig Mie, Morris Huw R, Pantazis Caroline B, Leonard Hampton L, Screven Laurel, Qi Yue A, Nalls Mike A, Bandres-Ciga Sara, Hardy John, Houlden Henry, Eng Celeste, Burchard Esteban González, Kachuri Linda, Lin Chia-Ho, Black Douglas L, Global Parkinson’s Genetics Program (GP2), Singleton Andrew B, Fischer Steffen, Bauer Peter, Reed Xylena, Ryten Mina, Beetz Christian, Ward Michael, Okubadejo Njideka U, Blauwendraat Cornelis

Аннотация

Recently, an African ancestry-specific Parkinson disease (PD) risk signal was identified at the gene encoding glucocerebrosidase (GBA1). This variant ( rs3115534 -G) is carried by ~50% of West African PD cases and imparts a dose-dependent increase in risk for disease. The risk variant has varied frequencies across African ancestry groups but is almost absent in European and Asian ancestry populations. GBA1 is a gene of high clinical and therapeutic interest. Damaging biallelic protein-coding variants cause Gaucher disease and monoallelic variants confer risk for PD and dementia with Lewy bodies, likely by reducing the function of glucocerebrosidase. Interestingly, the African ancestry-specific GBA1 risk variant is a noncoding variant, suggesting a different mechanism of action. Using full-length RNA transcript sequencing, we identified partial intron 8 expression in risk variant carriers (G) but not in nonvariant carriers (T). Antibodies targeting the N terminus of glucocerebrosidase showed that this intron-retained isoform is likely not protein coding and subsequent proteomics did not identify a shorter protein isoform, suggesting that the disease mechanism is RNA based. Clustered regularly interspaced short palindromic repeats editing of the reported index variant ( rs3115534 ) revealed that this is the sequence alteration responsible for driving the production of these transcripts containing intron 8. Follow-up analysis of this variant showed that it is in a key intronic branchpoint sequence and, therefore, has important implications in splicing and disease. In addition, when measuring glucocerebrosidase activity, we identified a dose-dependent reduction in risk variant carriers. Overall, we report the functional effect of a GBA1 noncoding risk variant, which acts by interfering with the splicing of functional GBA1 transcripts, resulting in reduced protein levels and reduced glucocerebrosidase activity. This understanding reveals a potential therapeutic target in an underserved and underrepresented population.

Предметные рубрики MeSH

ИП Урсолов Владлен Сергеевич

ИНН 500118482352 · ОГРНИП 314500117700049

Реквизиты и контакты