Neurobiology of aging · 2015
Discovery of gene-gene interactions across multiple independent data sets of late onset Alzheimer disease from the Alzheimer Disease Genetics Consortium.
СтатьяПоддержка NIH: внешняя программаПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод пока не подготовлен
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Hohman Timothy J, Bush William S, Jiang Lan, Brown-Gentry Kristin D, Torstenson Eric S, Dudek Scott M, Mukherjee Shubhabrata, Naj Adam, Kunkle Brian W, Ritchie Marylyn D, Martin Eden R, Schellenberg Gerard D, Mayeux Richard, Farrer Lindsay A, Pericak-Vance Margaret A, Haines Jonathan L, Thornton-Wells Tricia A, Alzheimer's Disease Genetics Consortium
Аннотация
Late-onset Alzheimer disease (AD) has a complex genetic etiology, involving locus heterogeneity, polygenic inheritance, and gene-gene interactions; however, the investigation of interactions in recent genome-wide association studies has been limited. We used a biological knowledge-driven approach to evaluate gene-gene interactions for consistency across 13 data sets from the Alzheimer Disease Genetics Consortium. Fifteen single nucleotide polymorphism (SNP)-SNP pairs within 3 gene-gene combinations were identified: SIRT1 × ABCB1, PSAP × PEBP4, and GRIN2B × ADRA1A. In addition, we extend a previously identified interaction from an endophenotype analysis between RYR3 × CACNA1C. Finally, post hoc gene expression analyses of the implicated SNPs further implicate SIRT1 and ABCB1, and implicate CDH23 which was most recently identified as an AD risk locus in an epigenetic analysis of AD. The observed interactions in this article highlight ways in which genotypic variation related to disease may depend on the genetic context in which it occurs. Further, our results highlight the utility of evaluating genetic interactions to explain additional variance in AD risk and identify novel molecular mechanisms of AD pathogenesis.
Предметные рубрики MeSH
ATP Binding Cassette Transporter, Subfamily BAlzheimer DiseaseCadherin Related ProteinsCadherinsCalcium Channels, L-TypeDatasets as TopicDisease ProgressionEpistasis, GeneticFemaleGenetic Association StudiesHumansMaleModels, GeneticPhosphatidylethanolamine Binding ProteinPolymorphism, Single NucleotideReceptors, Adrenergic, alpha-1Receptors, N-Methyl-D-AspartateRiskRyanodine Receptor Calcium Release ChannelSaposinsSirtuin 1