Neurobiology of aging · 2014
Late-onset Alzheimer's risk variants in memory decline, incident mild cognitive impairment, and Alzheimer's disease.
СтатьяПоддержка NIH: внешняя программаПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод пока не подготовлен
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Carrasquillo Minerva M, Crook Julia E, Pedraza Otto, Thomas Colleen S, Pankratz V Shane, Allen Mariet, Nguyen Thuy, Malphrus Kimberly G, Ma Li, Bisceglio Gina D, Roberts Rosebud O, Lucas John A, Smith Glenn E, Ivnik Robert J, Machulda Mary M, Graff-Radford Neill R, Petersen Ronald C, Younkin Steven G, Ertekin-Taner Nilüfer
Аннотация
We tested association of nine late-onset Alzheimer's disease (LOAD) risk variants from genome-wide association studies (GWAS) with memory and progression to mild cognitive impairment (MCI) or LOAD (MCI/LOAD) in older Caucasians, cognitively normal at baseline and longitudinally evaluated at Mayo Clinic Rochester and Jacksonville (n>2000). Each variant was tested both individually and collectively using a weighted risk score. APOE-e4 associated with worse baseline memory and increased decline with highly significant overall effect on memory. CLU-rs11136000-G associated with worse baseline memory and incident MCI/LOAD. MS4A6A-rs610932-C associated with increased incident MCI/LOAD and suggestively with lower baseline memory. ABCA7-rs3764650-C and EPHA1-rs11767557-A associated with increased rates of memory decline in subjects with a final diagnosis of MCI/LOAD. PICALM-rs3851179-G had an unexpected protective effect on incident MCI/LOAD. Only APOE-inclusive risk scores associated with worse memory and incident MCI/LOAD. The collective influence of the nine top LOAD GWAS variants on memory decline and progression to MCI/LOAD appears limited. Discovery of biologically functional variants at these loci may uncover stronger effects on memory and incident disease.
Предметные рубрики MeSH
ATP-Binding Cassette TransportersAgedAged, 80 and overAlzheimer DiseaseApolipoprotein E4ClusterinCognitive DysfunctionDisease ProgressionFemaleGenetic VariationGenome-Wide Association StudyHumansIncidenceMaleMembrane ProteinsMemoryMiddle AgedMonomeric Clathrin Assembly ProteinsReceptor, EphA1RiskWhite People