Archives des maladies du coeur et des vaisseaux · 2007
[Bi-tachycardia and ischemic cardiopathy (following myocardial infarction)].
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Marçon O, Brembilla-Perrot B
Аннотация
OBJECTIVE: The association of supraventricular tachycardia (SVT) and ventricular tachycardia (VT) is not common. The aim of this study was to evaluate its incidence and possible predisposing factors in patients with a history of myocardial infarction (MI), in order to correct them.
METHODS: Out of 359 patients consecutively recruited for spontaneous VT occurring at least one month after the acute phase of an MI, 33 were noted to have a possible association of SVT with VT. These patients underwent the following investigations: Holter recording, left ventricular ejection fraction (LVEF) measurement, and electrophysiological studies including programmed atrial and ventricular pacing.
RESULTS: The LEVF was 37.5+/-11%. An SVT was induced in 21 patients and a VT in 31 patients. Following investigation, the presumed factor predisposing to the two tachycardias was considered to be initiation of an SVT associated with rapid nodal conduction to the ventricle (Wenckebach point 210+/-23/mn) in 12 patients, branch to branch re-entry in 4 patients, reversible rhythmic dilated cardiomyopathy in 4 patients, an ischemic factor in 8 patients, post-operative period in 1 subject, infundibular VT in 1 subject, and an undetermined mechanism in 3 patients. The specific treatment of each of the identified etiologies usually prevented the recurrence of VT with the exception of one patient who had further VT during defibrillator recording, independent of any SVT.
CONCLUSION: The association of SVT with VT is rare because it was only noted in 9% of subjects who presented with spontaneous VT following myocardial infarction; This incidence is debatable, and could be an underestimate due to the fact that diagnosis is difficult. Several presumed predisposing factors are implicated and should be actively sought.
METHODS: Out of 359 patients consecutively recruited for spontaneous VT occurring at least one month after the acute phase of an MI, 33 were noted to have a possible association of SVT with VT. These patients underwent the following investigations: Holter recording, left ventricular ejection fraction (LVEF) measurement, and electrophysiological studies including programmed atrial and ventricular pacing.
RESULTS: The LEVF was 37.5+/-11%. An SVT was induced in 21 patients and a VT in 31 patients. Following investigation, the presumed factor predisposing to the two tachycardias was considered to be initiation of an SVT associated with rapid nodal conduction to the ventricle (Wenckebach point 210+/-23/mn) in 12 patients, branch to branch re-entry in 4 patients, reversible rhythmic dilated cardiomyopathy in 4 patients, an ischemic factor in 8 patients, post-operative period in 1 subject, infundibular VT in 1 subject, and an undetermined mechanism in 3 patients. The specific treatment of each of the identified etiologies usually prevented the recurrence of VT with the exception of one patient who had further VT during defibrillator recording, independent of any SVT.
CONCLUSION: The association of SVT with VT is rare because it was only noted in 9% of subjects who presented with spontaneous VT following myocardial infarction; This incidence is debatable, and could be an underestimate due to the fact that diagnosis is difficult. Several presumed predisposing factors are implicated and should be actively sought.