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Войти в исследования Retinal cases & brief reports · 2026
Foveal Micro-embolism in Subacute Bacterial Endocarditis and the Emerging Role of Neutrophil Extracellular Traps: A Case Report and Review of the Literature.
СтатьяНазвание и аннотация: Перевод пока не подготовлен
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Muste Justin C, Pulido Jose S, Fell David, Xu Timothy T, Zafar Sidra, Richards Collin J, Massenzio Erik, Aguwa Ugochi, Farrell Lily, Dunn James P, Deaner Jordan D
Аннотация
PURPOSE: To report a case of foveal micro-embolism in the setting of subacute bacterial endocarditis following routine dental cleaning.
METHODS: Retrospective case report and literature review.
RESULTS: A 45-year-old man presented with subacute bacterial endocarditis and a small distortion of vision his left eye. Aside from renal, splenic, and brain embolization the patient showed no splinter hemorrhages, Roth spots, Osler's nodes, and Janeway lesions. Further examination demonstrated a discrete, non-hemorrhagic lesion which corresponded to hyperreflective foci disrupting the inner and outer retinal layers on optical coherence tomography. This lesion resolved with observation and intravenous antibiotics.
DISCUSSION: We propose this case represents a distinct embolic mechanism mediated by neutrophil extracellular traps. Recognition of this mechanism may help expand our understanding of the spectrum of embolic phenomena in endocarditis.
METHODS: Retrospective case report and literature review.
RESULTS: A 45-year-old man presented with subacute bacterial endocarditis and a small distortion of vision his left eye. Aside from renal, splenic, and brain embolization the patient showed no splinter hemorrhages, Roth spots, Osler's nodes, and Janeway lesions. Further examination demonstrated a discrete, non-hemorrhagic lesion which corresponded to hyperreflective foci disrupting the inner and outer retinal layers on optical coherence tomography. This lesion resolved with observation and intravenous antibiotics.
DISCUSSION: We propose this case represents a distinct embolic mechanism mediated by neutrophil extracellular traps. Recognition of this mechanism may help expand our understanding of the spectrum of embolic phenomena in endocarditis.