International journal of cardiology · 2005
Supraventricular tachycardia promotes release of troponin I in patients with normal coronary arteries.
Письмо в редакциюНазвание и аннотация: Перевод пока не подготовлен
Обработка статьи · этапы, ошибки и управление
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История · было → стало
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Redfearn D P, Ratib K, Marshall H J, Griffith M J
Аннотация
BACKGROUND: Troponin I and T are sensitive markers of myocardial injury. The presence of elevated troponin often prompts further investigation with coronary angiography.
METHODS: We present a series of patients presenting to our unit with raised troponin I and supraventricular tachycardia (SVT) that prompted coronary angiography.
RESULTS: All patients had normal epicardial vessels at angiogram, no evidence of muscle bridging or pulmonary embolism.
CONCLUSION: The presence of tachycardia sufficient to warrant hospital admission can raise troponin, and this should be viewed in context when a decision on angiography is to be taken.
METHODS: We present a series of patients presenting to our unit with raised troponin I and supraventricular tachycardia (SVT) that prompted coronary angiography.
RESULTS: All patients had normal epicardial vessels at angiogram, no evidence of muscle bridging or pulmonary embolism.
CONCLUSION: The presence of tachycardia sufficient to warrant hospital admission can raise troponin, and this should be viewed in context when a decision on angiography is to be taken.