Nature cardiovascular research · 2024
A common gene signature of the right ventricle in failing rat and human hearts.
СтатьяНазвание и аннотация: Перевод пока не подготовлен
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Jurida Liane, Werner Sebastian, Knapp Fabienne, Niemann Bernd, Li Ling, Grün Dimitri, Wirth Stefanie, Weber Axel, Beuerlein Knut, Liebetrau Christoph, Wiedenroth Christoph B, Guth Stefan, Kojonazarov Baktybek, Jafari Leili, Weissmann Norbert, Günther Stefan, Braun Thomas, Bartkuhn Marek, Schermuly Ralph T, Dorfmüller Peter, Yin Xiaoke, Mayr Manuel, Schmitz M Lienhard, Czech Laureen, Schlüter Klaus-Dieter, Schulz Rainer, Rohrbach Susanne, Kracht Michael
Аннотация
The molecular mechanisms of progressive right heart failure are incompletely understood. In this study, we systematically examined transcriptomic changes occurring over months in isolated cardiomyocytes or whole heart tissues from failing right and left ventricles in rat models of pulmonary artery banding (PAB) or aortic banding (AOB). Detailed bioinformatics analyses resulted in the identification of gene signature, protein and transcription factor networks specific to ventricles and compensated or decompensated disease states. Proteomic and RNA-FISH analyses confirmed PAB-mediated regulation of key genes and revealed spatially heterogeneous mRNA expression in the heart. Intersection of rat PAB-specific gene sets with transcriptome datasets from human patients with chronic thromboembolic pulmonary hypertension (CTEPH) led to the identification of more than 50 genes whose expression levels correlated with the severity of right heart disease, including multiple matrix-regulating and secreted factors. These data define a conserved, differentially regulated genetic network associated with right heart failure in rats and humans.