Cell reports. Medicine · 2026
Syndecan-1-targeted therapeutic antibody impairs macropinocytosis and elicits antitumor immunity in pancreatic cancer.
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Yang Zecheng, Theardy Madelaine S, Chen Shuaitong, Wei Yongkun, Takeda Mitsunobu, Zeng Yue, Wang Xiaofei, Yao Jun, Li Jennifer, Thirasastr Prapassorn, Park Jangho, Zheng Yangxi, Vien Long T, Wani Khalida M, Wang Huamin, Gao Sisi, Heffernan Tim, Kwong Lawrence, Wistuba Ignacio I, Bover Laura, Draetta Giulio F, Ying Haoqiang, Yao Wantong
Аннотация
Pancreatic ductal adenocarcinoma (PDAC) remains one of the deadliest malignancies, with a 5-year survival rate of just 13%. While the development and early clinical use of small molecules targeting oncogenic KRAS mutations, key drivers of PDAC, have shown promise, resistance to these targeted therapies remains a significant challenge. We recently identified Syndecan-1 (SDC1), a highly expressed heparan sulfate proteoglycan, as a critical KRAS effector protein that promotes nutrient salvage and tumor growth. Here, we report the development of a human-specific monoclonal antibody (anti-SDC1 mAb) that inhibits PDAC cell proliferation in vitro and suppresses PDAC tumor growth in vivo. Mechanistically, the anti-SDC1 mAb blocks macropinocytosis and induces antibody-dependent cellular cytotoxicity (ADCC). In vivo, anti-SDC1 mAb synergizes with standard chemotherapy, KRAS∗ inhibitors, and immunotherapies, resulting in tumor regression and near-complete response. These findings highlight the anti-SDC1 mAb as a promising therapeutic strategy for PDAC and potentially other KRAS∗ and SDC1-driven tumors.