Steroids · 2026
Synthesis and cytotoxicity evaluation of steroidal 17β-hydroxy-17α-methylisoxazoles.
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Krech Anastasia, Aleksandrova Anna, Mekhtiev Arif, Timoshenko Olga, Kugaevskaya Elena, Zavialova Maria, Scherbakov Kirill, Chernysheva Anastasia, Sorokin Danila, Scherbakov Alexander, Tsybruk Tatsiana, Grabovec Irina, Zhabinskii Vladimir, Khripach Vladimir
Аннотация
The aim of this study was to develop an approach to the synthesis of 17β-hydroxy-17α-methylisoxazoles and to evaluate their cytotoxic effects against prostate cancer cell lines. The key reaction in their synthesis was the addition of a dianion generated from ethyl acetoacetate and butyllithium to androstenolone. Subsequent stages included the reaction of the intermediate β-hydroxyketone with hydroxylamine to form an isoxazole heterocycle, as well as manipulations with protecting groups. The cytotoxicity of the obtained compounds was studied using hormone-sensitive and hormone-independent prostate and breast carcinoma cell lines. It was found that 17β-hydroxy-17α-methylisoxazoles exerted a more pronounced inhibitory effect on LNCaP cells in comparison with abiraterone, while only minor cytotoxicity was observed toward PC3, MCF-7, and MDA-MB-231 cells. Analysis of the effects of the compounds on cell cycle progression in LNCaP cells demonstrated that they induced cell cycle arrest in the G0/G1 phase. Compound 4 (17α-(Isoxazol-3-ylmethyl)-androst-5-en-3β,17β-diol) suppressed the activity of key signaling pathways (AKT, S6K, ERKs) in LNCaP cells. Binding analysis of the studied compounds to human CYP17A1 revealed a weak spectral response, suggesting that effective binding does not occur in the active site and their mechanism of action is likely unrelated to CYP17A1 inhibition. The steroidal 17β-hydroxy-17α-methylisoxazoles are the basis for the development of new drugs that regulate the activity of kinase signaling pathways in tumor cells.