Journal of medicinal chemistry · 2024
Discovery of a Myeloid Cell Leukemia 1 (Mcl-1) Inhibitor That Demonstrates Potent In Vivo Activities in Mouse Models of Hematological and Solid Tumors.
СтатьяПоддержка NIH: внешняя программаПоддержка исследования: вне правительства СШАПоддержка правительства СШАНазвание и аннотация: Перевод пока не подготовлен
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Tarr James C, Salovich James M, Aichinger Martin, Jeon KyuOk, Veerasamy Nagarathanam, Sensintaffar John L, Arnhof Heribert, Samwer Matthias, Christov Plamen P, Kim Kwangho, Wunberg Tobias, Schweifer Norbert, Trapani Francesca, Arnold Allison, Martin Florian, Zhao Bin, Miriyala Nagaraju, Sgubin Danielle, Fogarty Stuart, Moore William J, Stott Gordon M, Olejniczak Edward T, Engelhardt Harald, Rudolph Dorothea, Lee Taekyu, McConnell Darryl B, Fesik Stephen W
Аннотация
Myeloid cell leukemia 1 (Mcl-1) is a key regulator of the intrinsic apoptosis pathway. Overexpression of Mcl-1 is correlated with high tumor grade, poor survival, and both intrinsic and acquired resistance to cancer therapies. Herein, we disclose the structure-guided design of a small molecule Mcl-1 inhibitor, compound 26, that binds to Mcl-1 with subnanomolar affinity, inhibits growth in cell culture assays, and possesses low clearance in mouse and dog pharmacokinetic (PK) experiments. Evaluation of 26 as a single agent in Mcl-1 sensitive hematological and solid tumor xenograft models resulted in regressions. Co-treatment of Mcl-1-sensitive and Mcl-1 insensitive lung cancer derived xenografts with 26 and docetaxel or topotecan, respectively, resulted in an enhanced tumor response. These findings support the premise that pro-apoptotic priming of tumor cells by other therapies in combination with Mcl-1 inhibition may significantly expand the subset of cancers in which Mcl-1 inhibitors may prove beneficial.