Cancer research · 2016
EGFL6 Regulates the Asymmetric Division, Maintenance, and Metastasis of ALDH+ Ovarian Cancer Cells.
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Bai Shoumei, Ingram Patrick, Chen Yu-Chih, Deng Ning, Pearson Alex, Niknafs Yashar S, O'Hayer Patrick, Wang Yun, Zhang Zhong-Yin, Boscolo Elisa, Bischoff Joyce, Yoon Euisik, Buckanovich Ronald J
Аннотация
Little is known about the factors that regulate the asymmetric division of cancer stem-like cells (CSC). Here, we demonstrate that EGFL6, a stem cell regulatory factor expressed in ovarian tumor cells and vasculature, regulates ALDH+ ovarian CSC. EGFL6 signaled at least in part via the oncoprotein SHP2 with concomitant activation of ERK. EGFL6 signaling promoted the migration and asymmetric division of ALDH+ ovarian CSC. As such, EGFL6 increased not only tumor growth but also metastasis. Silencing of EGFL6 or SHP2 limited numbers of ALDH+ cells and reduced tumor growth, supporting a critical role for EGFL6/SHP2 in ALDH+ cell maintenance. Notably, systemic administration of an EGFL6-neutralizing antibody we generated restricted tumor growth and metastasis, specifically blocking ovarian cancer cell recruitment to the ovary. Together, our results offer a preclinical proof of concept for EGFL6 as a novel therapeutic target for the treatment of ovarian cancer. Cancer Res; 76(21); 6396-409. ©2016 AACR.
Предметные рубрики MeSH
AdultAgedAged, 80 and overAldehyde DehydrogenaseAnimalsCalcium-Binding ProteinsCell Adhesion MoleculesCell DivisionCell Line, TumorExtracellular Signal-Regulated MAP KinasesFemaleHumansMembrane GlycoproteinsMiceMiddle AgedNeoplasm MetastasisNeoplastic Stem CellsOvarian NeoplasmsPrognosisProtein Tyrosine Phosphatase, Non-Receptor Type 11