Carotid and vagal afferents and drug action on transcallosally evoked cortical potentials.

The use of transcallosally evoked cortical potentials to study the action of intracarotidly injected drugs on cerebral synapses has necessitated the demonstration that vagal, baroreceptor, and chemoreceptor influences do not play essential roles in the drug effects observed-for example, the cerebral synaptic inhibitory action of serotonin.