Cancer research · 2002
Prevention of thymic lymphoma development in Atm-/- mice by dexamethasone.
СтатьяПоддержка исследования: вне правительства СШАПоддержка службы здравоохранения СШАНазвание и аннотация: Перевод пока не подготовлен
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Yan Mingshan, Kuang Xianghong, Qiang Wenan, Shen Jianjun, Claypool Kent, Lynn William S, Wong Paul K Y
Аннотация
We have reported (M. Yan et al., FASEB J., 15: 1132-1138, 2001) that spontaneous DNA synthesisis markedly increased in the thymocytes from the atrophied thymi of young Atm-/- mice. We, therefore, set out to determine whether this elevated DNA synthesis is responsible for the development of thymic lymphomas in all Atm-/- mice by 4-5 months of age. We show here that in Atm-/- mice: (a) increased DNA synthesis occurs, especially in the immature CD4(-) CD8(-) (dominant negative) and CD8(+) thymocyte populations; (b) the relative percentage of dominant negative cells increases significantly during postnatal development, with a sharp peak at 4 weeks of age; and (c) dexamethasone suppresses DNA synthesis in these thymocytes and prevents thymic lymphoma development. These observations suggest that ataxia telangiectasia mutated (ATM) down-regulates the proliferation of thymocytes, allowing T-cell development and differentiation. The results also show that dexamethasone, like ATM, checks DNA synthesis in developing thymocytes. Finally, the data document for the first time that dexamethasone prevents or slows thymic lymphoma development in Atm-/- mice.
Предметные рубрики MeSH
AnimalsAnticarcinogenic AgentsApoptosisAtaxia Telangiectasia Mutated ProteinsCell Cycle ProteinsCell DivisionDNADNA-Binding ProteinsDexamethasoneDown-RegulationFemaleLymphomaMaleMiceNucleic Acid Synthesis InhibitorsProtein Serine-Threonine KinasesThymus GlandThymus NeoplasmsTumor Suppressor Proteins