Journal of immunology (Baltimore, Md. : 1950) · 2005
The prevalence of regulatory T cells in lymphoid tissue is correlated with viral load in HIV-infected patients.
СтатьяПоддержка исследования: вне правительства СШАПоддержка службы здравоохранения СШАНазвание и аннотация: Перевод пока не подготовлен
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Andersson Jan, Boasso Adriano, Nilsson Jakob, Zhang Rui, Shire Norah J, Lindback Stefan, Shearer Gene M, Chougnet Claire A
Аннотация
Inadequate local cell-mediated immunity appears crucial for the establishment of chronic HIV infection. Accumulation of regulatory T cells (Treg) at the site of HIV replication, the lymphoid organs, may influence the outcome of HIV infection. Our data provide the first evidence that chronic HIV infection changes Treg tissue distribution. Several molecules characteristics of Treg (FoxP3, CTLA-4, glucocorticoid-induced TNFR family-related receptor, and CD25) were expressed more in tonsils of untreated patients compared with antiretroviral-treated patients. Importantly, most FoxP3+ cells expressed CTLA-4, but not CD69. Furthermore, a direct correlation between FoxP3 levels and viral load was evident. In contrast, FoxP3 expression was decreased in circulating T cells from untreated patients, but normalized after initiation of treatment. Functional markers of Treg activity (indoleamine 2,3-dioxygenase, TGF-beta, and CD80) were markedly increased in the tonsils of untreated patients. Our data could provide a new basis for immune-based therapies that counteract in vivo Treg and thereby reinforce appropriate antiviral immunity.
Предметные рубрики MeSH
AdultAntigens, CDAntigens, DifferentiationAntiretroviral Therapy, Highly ActiveCTLA-4 AntigenDNA-Binding ProteinsForkhead Transcription FactorsGlucocorticoid-Induced TNFR-Related ProteinHIV InfectionsHIV-1HumansLymphoid TissuePalatine TonsilRNA, MessengerReceptors, Interleukin-2Receptors, Nerve Growth FactorReceptors, Tumor Necrosis FactorT-LymphocytesTransforming Growth Factor betaTransforming Growth Factor beta1