Nature communications · 2018
Genetic alterations driving metastatic colony formation are acquired outside of the primary tumour in melanoma.
СтатьяПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод пока не подготовлен
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Werner-Klein Melanie, Scheitler Sebastian, Hoffmann Martin, Hodak Isabelle, Dietz Klaus, Lehnert Petra, Naimer Veronika, Polzer Bernhard, Treitschke Steffi, Werno Christian, Markiewicz Aleksandra, Weidele Kathrin, Czyz Zbigniew, Hohenleutner Ulrich, Hafner Christian, Haferkamp Sebastian, Berneburg Mark, Rümmele Petra, Ulmer Anja, Klein Christoph A
Аннотация
Mouse models indicate that metastatic dissemination occurs extremely early; however, the timing in human cancers is unknown. We therefore determined the time point of metastatic seeding relative to tumour thickness and genomic alterations in melanoma. Here, we find that lymphatic dissemination occurs shortly after dermal invasion of the primary lesion at a median thickness of ~0.5 mm and that typical driver changes, including BRAF mutation and gained or lost regions comprising genes like MET or CDKNA2, are acquired within the lymph node at the time of colony formation. These changes define a colonisation signature that was linked to xenograft formation in immunodeficient mice and death from melanoma. Thus, melanoma cells leave primary tumours early and evolve at different sites in parallel. We propose a model of metastatic melanoma dormancy, evolution and colonisation that will inform direct monitoring of adjuvant therapy targets.