EMBO reports · 2026
Interspecies regulation of virulence through gingipain inhibition by a Tannerella forsythia serpin.
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Książek Mirosław, Benedyk-Machaczka Małgorzata, Mizgalska Danuta, Sochaj-Gregorczyk Alicja, Waligórska Irena, Drzazga Kamila, Nowak Magdalena, Kaczor Anna, Potempa Barbara, Fuchs Andrew, Thøgersen Ida B, Enghild Jan J, Scavenius Carsten, Mydel Piotr, Moris Savannah C, Bagaitkar Juhi, Potempa Jan
Аннотация
Periodontitis is driven by dysbiotic microbial communities in which Porphyromonas gingivalis (Pg) acts as a keystone pathogen through its caspase-like fold (Clan CD) proteases, gingipains. Tannerella forsythia (Tf), another member of the periodontal pathogenic community, uniquely produces the broad-spectrum serpin termed miropin, yet its biological significance as an inhibitor in periodontal inflammation remains unclear. Here, we demonstrate that miropin directly inhibits the lysine-specific gingipain Kgp and the calpain-like protease Tpr produced by Pg, forming irreversible covalent inhibitory complexes. While recombinant native miropin impairs Pg growth, an engineered variant targeting both lysine- and arginine-specific gingipains entirely abolishes bacterial growth in a defined medium. In vivo, recombinant miropin reduces Pg virulence and, when co-infected with wild-type Tf, prevents lethal infection and significantly reduces alveolar bone loss, but not when co-infected with a miropin-deficient mutant. Protection is associated with inhibition of gingipain activity and normalization of the RANKL/OPG ratio in periodontal tissues. These findings identify miropin as a potent interspecies virulence modulator and challenge the prevailing view of Tf as an exclusively pathogenic member of the oral microbiome.