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DOI 10.1016/j.jjcc.2017.11.010

Медицинская библиотека · Journal of cardiology · 2017

Late onset of atrial fibrillation in patients undergoing mitral valve repair for type II dysfunction.

Kawamoto Naonori, Fujita Tomoyuki, Fukushima Satsuki, Shimahara Yusuke, Kume Yuta, Matsumoto Yorihiko, Yamashita Kizuku, Kobayashi Junjiro

Оценочное исследованиеСтатьяПеревод в очереди

Аннотация

BACKGROUND

There are few reports regarding factors related to late-onset of atrial fibrillation (Af) after mitral valve repair, which can compromise long-term outcome. We analyzed the risk factors for late Af after mitral valve repair.

METHODS AND

RESULTS

Between 2001 and 2015, 318 patients (214 males; median, 57.7 years) underwent mitral valve repair for degenerative mitral insufficiency (MI) without concomitant aortic valve disease in our institute. Patients with a history of paroxysmal or chronic Af preoperatively were excluded. Serial follow-up echocardiography was used to evaluate cardiac function. The follow-up rate was 99%. The mean follow-up period was 6.0±4.1 years. There was no early death and there were nine late deaths. A total of 29 (9.1%) patients developed late Af. Freedom from late Af was 94.0% at 5 years and 82.9% at 10 years. Small ring annuloplasty, left atrial diameter, and pressure half time, which were measured at follow-up echocardiography, were considered as significant (p<0.05) risk factors for late Af. Patients who had late Af developed recurrent MI more frequently than those who did not (p<0.05).

CONCLUSIONS

Remodeling of the atrium caused by functional mitral stenosis is important for late Af, as well as recurrent MI. To prevent these problems, avoiding selection of a small ring at primary mitral repair for type II dysfunction and durable repair are required.

Для цитирования: Kawamoto Naonori, Fujita Tomoyuki, Fukushima Satsuki, Shimahara Yusuke, Kume Yuta, Matsumoto Yorihiko, Yamashita Kizuku, Kobayashi Junjiro. Late onset of atrial fibrillation in patients undergoing mitral valve repair for type II dysfunction.. Journal of cardiology. 2017. DOI: 10.1016/j.jjcc.2017.11.010.

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