Nature communications · 2013
Heterozygous mutations in PALB2 cause DNA replication and damage response defects.
СтатьяПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
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Nikkilä Jenni, Parplys Ann Christin, Pylkäs Katri, Bose Muthiah, Huo Yanying, Borgmann Kerstin, Rapakko Katrin, Nieminen Pentti, Xia Bing, Pospiech Helmut, Winqvist Robert
Аннотация
Besides mutations in BRCA1/BRCA2, heterozygous defects in PALB2 are important in breast cancer predisposition. PALB2 heterozygosity increases the risk of malignancy about sixfold. PALB2 interacts with BRCA1 and BRCA2 to regulate homologous recombination and mediate DNA damage response. Here we show, by analysing lymphoblastoid cell lines from heterozygous female PALB2 mutation carriers, that PALB2 haploinsufficiency causes aberrant DNA replication/damage response. Mutation carrier cells show increased origin firing and shorter distance between consecutive replication forks. Carrier cell lines also show elevated ATR protein, but not phosphorylation levels, and a majority of them display aberrant Chk1-/Chk2-mediated DNA damage response. Elevated chromosome instability is observed in primary blood lymphocytes of PALB2 mutation carriers, indicating that the described mechanisms of genome destabilization operate also at the organism level. These findings provide a new mechanism for early stages of breast cancer development that may also apply to other heterozygous homologous recombination signalling pathway gene mutations in hereditary cancer predisposition.
Предметные рубрики MeSH
Ataxia Telangiectasia Mutated ProteinsBRCA1 ProteinBRCA2 ProteinBreast NeoplasmsCase-Control StudiesCell Line, TumorCheckpoint Kinase 1Checkpoint Kinase 2Chromosomal InstabilityDNA DamageDNA ReplicationFanconi Anemia Complementation Group N ProteinFemaleGene Expression Regulation, NeoplasticGenetic Predisposition to DiseaseHaploinsufficiencyHeterozygoteHumansLymphocytesMutationNuclear ProteinsPrimary Cell CultureProtein KinasesTumor Suppressor Proteins