Genetics · 2025
Revealing function-altering MECP2 mutations in individuals with autism spectrum disorder using yeast and Drosophila.
СтатьяПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
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Chen Eric, Schmitt Jessica, McIntosh Graeme, Young Barry P, Lian Tianshun, Liu Jie, Chen Kexin K, Liston J Beatrice, MacDonald Lily, Wang Bill, Medina Giro Sonia, Boehme Benjamin, Das Mriga, Indran Seevasant, Chao Jesse T, Rogic Sanja, Pavlidis Paul, Allan Douglas W, Loewen Christopher J R
Аннотация
Pathogenic variants in MECP2 commonly lead to Rett syndrome, where MECP2's function as a DNA cytosine methylation reader is believed critical. MECP2 variants are also cataloged in individuals with autism spectrum disorder (ASD), including nine missense variants which had no known clinical significance at the start of this study. To assess these nine variants as risk alleles for ASD, we developed MECP2 variant functional assays using budding yeast and Drosophila. We calibrated these assays with known pathogenic and benign variants. Our data predict that four ASD variants are loss of function and five are functional. Protein destabilization offers insight into the altered function of some of these variants. Notably, yeast and Drosophila lack DNA methylation, yet all Rett pathogenic and ASD variants located in the methyl DNA-binding domain that we analyzed proved to be loss of function, suggesting a clinically relevant role for non-methyl DNA-binding by MECP2.