American journal of human genetics · 2013
Mutations in PDGFRB cause autosomal-dominant infantile myofibromatosis.
СтатьяПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
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Martignetti John A, Tian Lifeng, Li Dong, Ramirez Maria Celeste M, Camacho-Vanegas Olga, Camacho Sandra Catalina, Guo Yiran, Zand Dina J, Bernstein Audrey M, Masur Sandra K, Kim Cecilia E, Otieno Frederick G, Hou Cuiping, Abdel-Magid Nada, Tweddale Ben, Metry Denise, Fournet Jean-Christophe, Papp Eniko, McPherson Elizabeth W, Zabel Carrie, Vaksmann Guy, Morisot Cyril, Keating Brendan, Sleiman Patrick M, Cleveland Jeffrey A, Everman David B, Zackai Elaine, Hakonarson Hakon
Аннотация
Infantile myofibromatosis (IM) is a disorder of mesenchymal proliferation characterized by the development of nonmetastasizing tumors in the skin, muscle, bone, and viscera. Occurrence within families across multiple generations is suggestive of an autosomal-dominant (AD) inheritance pattern, but autosomal-recessive (AR) modes of inheritance have also been proposed. We performed whole-exome sequencing (WES) in members of nine unrelated families clinically diagnosed with AD IM to identify the genetic origin of the disorder. In eight of the families, we identified one of two disease-causing mutations, c.1978C>A (p.Pro660Thr) and c.1681C>T (p.Arg561Cys), in PDGFRB. Intriguingly, one family did not have either of these PDGFRB mutations but all affected individuals had a c.4556T>C (p.Leu1519Pro) mutation in NOTCH3. Our studies suggest that mutations in PDGFRB are a cause of IM and highlight NOTCH3 as a candidate gene. Further studies of the crosstalk between PDGFRB and NOTCH pathways may offer new opportunities to identify mutations in other genes that result in IM and is a necessary first step toward understanding the mechanisms of both tumor growth and regression and its targeted treatment.